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by Georg E. Cold

Download Cerebral Blood Flow in Acute Head Injury: The Regulation of Cerebral Blood Flow and Metabolism During the Acute Phase of Head Injury, and Its Significance for Therapy (Acta Neurochirurgica Supplement) ePub
  • ISBN 3211822240
  • ISBN13 978-3211822241
  • Language English
  • Author Georg E. Cold
  • Publisher Springer; 1 edition (October 16, 2003)
  • Pages 64
  • Formats rtf lrf lit doc
  • Category Medicine
  • Subcategory Medicine
  • Size ePub 1338 kb
  • Size Fb2 1503 kb
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During the last decade a multitude of studies concerning the dynamic changes in cerebral blood flow (CBF), cerebral metabolic rate of oxygen (CMRO2), and intracranial pressure (ICP) in the acute phase after head injury have been published. These studies have been supplemented with studies of cerebral autoregulation, CO2 reactivity and barbiturate reactivity. Other investigations include studies of cerebrospinal fluid pH, bicarbonate, lactate and pyruvate. In this book experimental and clinical studies of the dynamic changes in CBF, CMRO2, CO2 reactivity and barbiturate reactivity are reviewed. The author's own clinical studies of the dynamic changes in CBF and cerebral metabolism are summarized and discussed, and the therapeutical implication as regards the use of artificial hyperventilation, sedation with barbiturate and mannitol treatment are discussed.

During the last decade a multitude of studies concerning the dynamic changes in cerebral blood flow (CBF) . These studies have been supplemented with studies of cerebral autoregulation, CO2 reactivity and barbiturate reactivity

During the last decade a multitude of studies concerning the dynamic changes in cerebral blood flow (CBF), cerebral metabolic rate of oxygen (CMRO2), and intracranial pressure (ICP) in the acute phase after head injury have been published. These studies have been supplemented with studies of cerebral autoregulation, CO2 reactivity and barbiturate reactivity. Other investigations include studies of cerebrospinal fluid pH, bicarbonate, lactate and pyruvate.

We aim to show you accurate product information. Manufacturers, suppliers and others provide what you see here, and we have not verified it. See our disclaimer. Cerebral blood flow in acute head injury. ACTA Neurochirurgica Supplement. Springer Vienna, Springer.

During the last decade several studies of cerebral blood flow (CBF) and metabolism in the acute phase of head injury have been published. Published: 1 January 1990. in Acta neurochirurgica.

oceedings{Cold1990CerebralBF, title {Cerebral Blood Flow in Acute Head Injury}, author {Georg Emil . Cerebral blood flow and metabolism in severely head-injured children

oceedings{Cold1990CerebralBF, title {Cerebral Blood Flow in Acute Head Injury}, author {Georg Emil Cold}, booktitle {Acta Neurochirurgica}, year {1990} }. Georg Emil Cold. Published in Acta Neurochirurgica 1990. Cerebral blood flow and metabolism in severely head-injured children. Part I: Relationship with GCS score, outcome, ICP, and PVI. A Marmarou, AAF DeSalles, +4 authors HF Young. Effect of hypocapnia on local cerebral glucose utilization in rats. Satwant K. Samra, P. Turk, James F. Arens.

Following severe head injury, derangements of the cerebral vasculature . In supratentorial cortical lesions, an acute fall in Paco2 increased the homogeneity of the regional flow pattern (decrease in th. .

Following severe head injury, derangements of the cerebral vasculature and cerebral blood flow (CBF) often occur, rendering the brain at risk of secondary ischemia. Regulation of cerebral blood flow is complex and differs from regulation of blood flow in other vascular beds. Basic vascular tone formed by interaction of vascular smooth muscle cells and endothelium provides background for other regulatory mechanisms. In supratentorial cortical lesions, an acute fall in Paco2 increased the homogeneity of the regional flow pattern (decrease in the standard deviation of the regional flow values), and reduced the number of focal hyperaemic regions (tissue peaks).

Putative pathways regulating cerebral blood flow during hypoxia. Increased temperature, erythrocyte deformation, and the conformational change concomitant to transition of oxy- to deoxyhemoglobin all signal erythrocyte mediated release of ATP (,,, ). Released ATP can then bind to the erythrocyte P2X7 receptor in an autocrine fashion to induce erythrocyte mediated EET release (), which will increase vascular smooth muscle cell K+ channel conductance ().

Beyond intracranial pressure: optimization of cerebral blood flow 31. Danura H. Acute Angiographic Vasospasm and the Incidence of Delayed Cerebral Vasospasm in Preliminary Results.

Beyond intracranial pressure: optimization of cerebral blood flow. oxygen and substrate delivery after traumatic brain injury // Annals of Intensive Care. Vol. 10 № 3. Р. 23. doi: 1. 186/2110-5820-3-23. The dynamics of cerebral oxygenation and blood flow in response to changes in arterial pressure and intracranial pressure after head injury // Br. J. Anaesth. 108. № 1. P. 89-99. 31. Acute Angiographic Vasospasm and the Incidence of Delayed Cerebral Vasospasm in Preliminary Results, Neurovascular Events After Subarachnoid Hemorrhage // Acta Neurochirurgica Supplement.

According to the Journal Citation Reports, the journal has a 2018 impact factor of . 40.

Cerebral Blood Flow and Its Regulation after Closed Head Injury with Emphasis on Clinical Correlations, Journal of Neurosurgery, Vol. 41, No. 5, 1974, pp. 531-541. W. D. Obrist, T. Langfitt, J. L. Jaggi, J. Cruz and T. A. Gennarelli, Cerebral Blood Flow and Metabolism in Comatose Patients with Acute Head Injury. Relationship to Intracranial Hypertension, Journal of Neurosurgery, Vol. 61, No. 2, 1984, pp. 241-253.

Department of Traumatology and Acute Critical Medicine, Osaka University Graduate School of Medicine, 2-15 Yamadaoka, Suita, Osaka 565-0871, Japan

Department of Traumatology and Acute Critical Medicine, Osaka University Graduate School of Medicine, 2-15 Yamadaoka, Suita, Osaka 565-0871, Japan.